Being Healthy Is Unprofitable

healthcareCharles Hugh Smith – That good health is insanely unprofitable was highlighted by a staggering statistic in the recent research paper The Concentration of Health Care Spending (via B.C.):

Mean annual spending for the bottom half of (the American population) distribution was just $236 per person, totaling only $36 billion for the entire group of more than 150 million people.

We don’t know why the 150 million people did not consume much in the way of “health services”– they might have been healthy and had no need for healthcare beyond routine tests, or they might have needed care and been unable to afford it, despite the Orwellian-titled Affordable Care Act (ACA).

But let’s assume that the 150 million people–roughly half of America’s 317 million residents–were healthy and had no need for health services beyond minimal prevention and a few low-cost tests.

The total cost of their care was $36 billion–just over 1% of the nation’s $3.2 trillion bill for healthcare and healthcare insurance. Let’s assume that 90% of the populace was healthy, and the remaining 10% were very ill and needed 100 times as much care as the healthy.

The total cost of caring for the 285 million healthy people would be roughly $67 billion, or just over 2% of the $3 trillion we currently spend on healthcare. The very ill 32 million would need $23,600 each, or $755 billion.

The total cost for a largely healthy population and 32 million ill people who required 100 times more care than the healthy would be $822 billion, or roughly 25% of the $3.2 trillion we currently spend annually. Continue reading

Eat More Healthy Fats. Here’s Why & How You Can Revolutionize Your Health

Increasing the amount of high quality fat, including saturated fat, in your diet may be the best thing you can do for your health. It’s time to put aside the low-fat diet schemes that have dominated the diet industry for over 40 years.

Sound, scientific research supports the reasonable consumption of healthy fats, including saturated fat, as part of a healthy lifestyle. This article takes a brief look at how we lost our way, what science now says about dietary fats, and which foods may be safely added to our diets.

How Did We Get Here?

In 1977 the USDA, through the National Advisory Committee on Nutritional Education (NACNE), recommended that Americans:

1 – Reduce total dietary fat to 30%

2 – Reduce saturated fat to 10% of total calories.

These recommendations were made without scientific, randomized controlled trials (RCTs) being performed to test their validity before being implemented. Furthermore, there were only five randomized trials of unhealthy men (no women) available to the committee at the time.  The committee was also heavily influenced by the now controversial and partially discredited Seven Countries Study of Ancel Keys which implicated saturated fat in cardiovascular disease.

Significantly, the rise in obesity corresponds with the publication of the government’s dietary standards:

Obesity

 

New Research

A recent meta-review of the 1977 recommendations appeared in the prestigious British Medical Journal’s OpenHeart and put them to rest. The authors, Harcombe, et al, concluded:

  • “It seems incomprehensible that dietary recommendations were introduced to 220 million US and 56 million UK citizens given the contrary results from a small number of unhealthy men.”
  • “The results of the present meta-analysis support the hypothesis that the available RCTs did not support the introduction of dietary fat recommendations in order to reduce CHD risk or related mortality.”
  • And that the dietary recommendations  “should not have been introduced.”

Implementing these dietary measures has been devastating. According to the CDC: “Between 1980 and 2000, obesity rates doubled among adults. About 60 million adults, or 30% of the adult population, are now obese.” Obesity rates continue to soar with Type 2 diabetes now at epidemic proportions.

What Does Current Nutrition Science Say About Saturated Fat?   

Much of current research paints a very different picture of the role of saturated fat in our diets. Let’s look briefly at four major studies that represent current thinking about dietary fat and specifically saturated fat.

Study 1 – The first study is a retrospective look at a trial completed in the early 1970s, the data from which had been lost. Dr. Christopher E. Ramden led an Australian and US team to evaluate “recovered data from the Sydney Diet Heart Study, a single blinded, parallel group, randomized controlled trial conducted in 1966-73; and an updated meta-analysis including these previously missing data.” The original study attempted to evaluate the effectiveness of replacing saturated fat with omega 6 linoleic acid, a vegetable oil. Participants included 458 men aged 30-59 who had experienced a recent coronary event.

Conclusion: “In this cohort, substituting dietary linoleic acid in place of saturated fats increased the rates of death from all causes, coronary heart disease, and cardiovascular disease. An updated meta-analysis of linoleic acid intervention trials showed no evidence of cardiovascular benefit.”

Study 2 – In 2010 a meta-analysis of 21 prospective studies evaluated the association of saturated fat and cardiovascular disease. The results were published in the American Journal of Clinical Nutrition and led by Patty W Siri-Tarino of the Children’s Hospital Oakland Research Institute.

Conclusion: “A meta-analysis of prospective epidemiologic studies showed that there is no significant evidence for concluding that dietary saturated fat is associated with an increased risk of CHD or CVD.” In other words, there is no verifiable link between eating saturated fat and the occurrence of coronary heart disease.

Study 3Cambridge scholar, Dr. Chowdhury, and an international research team in 2014, evaluated nearly 80 studies, including 27 randomized controlled trials (RCTs, the gold standard of scientific research) that involved half a million people. The research included not only what people reported they ate but measured the composition of fatty acids in their blood and fatty tissues.

Conclusion: The researchers found that “…current evidence does not support guidelines which restrict the consumption of saturated fats in order to prevent heart disease.” According to the NY Times: “The researchers did find a link between trans fats… and heart disease but ‘they found no evidence of dangers from saturated fat, or benefits from other kinds of fats.’”

Study 4 – In 2014, Dr. Jeff Volek, a professor of Human Sciences at Ohio State University, and his research team, recruited 16 adults, all of whom suffered from metabolic syndrome. Participants were fed diets that changed every three weeks up to 18 weeks. Every three weeks the amount of saturated fat was decreased and the amount of carbohydrates increased, and the amount of palmitoleic acid in the blood was measured. Palmitoleic acid has been linked to obesity, inflammation, insulin resistance, glucose intolerance, type 2 diabetes, heart disease and prostate cancer.

Conclusion: “When looking at palmitoleic acid… the scientists found that it consistently decreased on the high-fat/low-carb diet in all participants. The fatty acid then showed a step-wise increase in concentration in the blood as carbs were progressively added to the diet.”

In other words, as carbohydrates were added to the diet, levels of the deadly palmitoleic acid increased, thus heightening the risk of CVD.

Dr. Volek concluded: “There is widespread misunderstanding about saturated fat… there’s clearly no association of dietary saturated fat and heart disease, yet dietary guidelines continue to advocate restriction of saturated fat.”

What Have We Learned from These Scientific Studies?

  1. The 1977 dietary recommendations to limit saturated fat were not scientifically valididated
  2. There is no clear association between saturated fat and heart disease
  3. Trans fats (found in processed meats and foods, and vegetable oils) are linked to increased cardiovascular disease
  4. High fat/low carbohydrate diets lower dangerous levels of palmitoleic acid, which is associated with heart disease and other chronic diseases
  5. High levels of Omega 6 fatty acids in the form of linoleic vegetable oil showed no positive cardiovascular benefit.

What High-Fat Foods Should We Be Eating?

The following foods, some high in saturated fat, are healthy to consume as part of a natural, whole foods diet:

Avocado, tahini, dark chocolate, eggs, fatty fish, nuts, coconut, and liver. When combined with low carbohydrate foods, (spinach, broccoli, cauliflower, carrots, and others) coconut and olive oil, fresh fruits in moderation, and meat (preferably grass fed organic), a wholesome and healthy diet that guards against heart and other chronic diseases can be attained. Keep in mind that calories do count. It behooves us to eat reasonable portions as well as reduce carbohydrate levels in our diets. It’s both what we eat and how much we eat that matters.

*These foods are recommended given we are all driven towards various diet types and making healthier choices in each diet type is a step in the right direction.

A complete meal plan based on low carbohydrate and healthy fat intake can be found at Authority Nutrition.

Conclusion

Deciding on a diet best for you should be done with an awareness of current scientific thinking and in consultation with your physician or health provider. One diet does not fit all. The best diet for each of us is one that meets our individual needs.

The information in this article is not meant as medical advice and should be used for educational purposes only.


SF Source CollectiveEvolution  Feb 22 2015

Junk Food More Deadly Than War, Famine, Genocide

JunkFoodDietHealth“Food in the end, in our tradition, is something holy. It’s not about nutrients and calories. It’s about sharing. It’s about honesty. It’s about identity.” ~ Louise Fresco

You may already know that junk food is bad for your health, but you may not realize how bad it can be. A new study from the School of Medical Sciences at Australia’s University of New South Wales points to profound brain changes that junk food causes, making a junk food habit “more deadly than war, famine, and genocide”.

Say what? Yep, the food war is real, and though the UNSW study was conducted on rats, the brain changes observed matter to us humans. As mammals we share similar brain functioning in the orbitofrontal cortex, the part of our gray matter responsible for sensing and evaluating the pleasurable aspects of food.

Makers of junk food know it is highly addictive, but the UNSW study proves unequivocally that junk food alters behavior by causing near-permanent changes in the brain’s reward circuiting, an alteration that can trigger obesity.

The study abstract concluded:

“We observed that rats fed a cafeteria diet for 2 weeks showed impaired sensory-specific satiety following consumption of a high calorie solution. The deficit in expression of sensory-specific satiety was also present 1 week following the withdrawal of cafeteria foods. Thus, exposure to obesogenic diets may impact upon neurocircuitry involved in motivated control of behavior.”

While mammals developed a natural trigger over our evolutionary history which prevents us from over-eating, a phenomenon termed “sensory-specific satiety,” the consumption of junk food overrides this natural ‘kill’ switch that allows us to regulate the calories we consume. Continue reading

Blueberry Phenol Pterostilbene Reduces Body Fat, Could Lower Diabetes Risk

“. . . a 2003 study found that short-lived fish given resveratrol lived 50 percent longer and both learned and swam better into old age.” – D Gutierrez

BlueberriesNaturalNews – You may have heard about the miracle antioxidant resveratrol, found in blueberries, grapes and red wine. But did you know that these same foods also contain another phenolic compound, pterostilbene, which may help lower body fat and reduce diabetes risk? That was the finding of a study conducted by researchers from the Spanish Biomedical Research Centre in Physiopathology of Obesity and Nutrition (CIBERobn) of the Carlos III Institute of Health, in collaboration with the U.S. Department of Agriculture.

Pterostilbene is in the same chemical family as resveratrol and is found in many of the same foods, such as grapes, blueberries, cranberries and peanuts. Pterostilbene is much less well studied than resveratrol, and most studies to date have focused on its cancer-fighting benefits. Because of its small size, pterostilbene is able to penetrate the cell membranes of cancer cells, inducing cell death. It has also been shown to suppress the ability of cancer cells to produce the energy they need to function.

The new study is the first pre-clinical work to look at pterostilbene’s effects on obesity in an animal model.

Prevents diabetes, protects heart

Continue reading

Food Addiction And The Obesity Epidemic

Wake Up World  February 19 2014

Have you ever heard someone describe a certain food as addictive? Of course you have. Certainly when we eat specific foods, it feels like we can’t seem to get enough. And we also have a tendency to turn to those foods for emotional comfort.

We also understand that binge eating is considered an eating disorder – it is categorized as a mental-emotional disorder in the Diagnostic and Statistical Manual of Mental Disorders (DSM-5). However, the bigger question is whether binge eating is addictive? And is food addiction contributing to the obesity epidemic?

Food addiction is real

According to Dr. S. Dickson, it is important to understand the mechanisms underlying the uncontrolled intake of food and the development of obesity. She remarked that “brain reward pathways that are involved in alcohol and drug addiction are also essential elements of the ghrelin responsive circuit. And ghrelin has been shown to both signal hunger and increase food intake”.

Ghrelin is a hormone secreted by the stomach and pancreas which stimulates areas of the brain responsible for hunger – and it might be responsible for food addiction, making some people more prone to obesity.

Dr. Dickson also added, “based on these and other recent findings, could obesity be a food addiction? A subgroup of obese patients indeed show ‘addictive-like’ properties with regard to overeating…. but this does not automatically mean they are addicted”.

“We don’t completely understand why certain vulnerable individuals become addicted, transferring something rewarding to becoming addicted to it,” she noted. “For now, we need to ask: in our modern environment where food is so plentiful, has food no longer become our friend when it is something we can become addicted to?”

Continue reading